The frequency of loud sound exposure affects the risk of developing tinnitus
Evidence from both animal models and human population studies robustly supports the conclusion that exposure to loud sounds or acoustic trauma increases the risk of developing tinnitus.
The retrieved papers consistently support the claim that loud sound or noise exposure is a major risk factor for developing tinnitus. Papers [0], [1], [6], and [9] use acoustic trauma to successfully model and study the onset of tinnitus in animals. Paper [7] demonstrates a clear dose-response relationship between occupational noise levels and tinnitus risk in humans. Other papers (such as [2], [5], [8]) discuss tinnitus in the context of noise-exposed musicians. Therefore, the balance of evidence strongly supports the claim.
M. Guitton, Y. Dudai. Blockade of Cochlear NMDA Receptors Prevents Long-Term Tinnitus during a Brief Consolidation Window after Acoustic Trauma. 2008. https://doi.org/10.1155/2007/80904
This study demonstrates that acoustic trauma (loud sound exposure) triggers long-term tinnitus in an animal model, which can be prevented by blocking NMDA receptors.
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T. Brozoski, Kurt W. Wisner, L. Sybert, C. Bauer. Bilateral Dorsal Cochlear Nucleus Lesions Prevent Acoustic-Trauma Induced Tinnitus in an Animal Model. 2011. https://doi.org/10.1007/s10162-011-0290-3
This study shows that high-level noise exposure induces tinnitus in rats, and that specific neural lesions can prevent this noise-induced outcome.
Kate Lewkowski, J. Heyworth, W. Williams, Helen Goulios, K. McCausland, Corie Gray, L. Fritschi. The Associations Between Workplace Noise, Ototoxic Chemicals, and Tinnitus. 2023. https://doi.org/10.1097/AUD.0000000000001392
This population-level study finds a clear dose-response relationship between occupational noise exposure above legal limits and an increased risk of developing tinnitus.
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