Unilateral damage to the vagus nerve produces specific physiological deficits

the verdict

SUPPORTED

the evidence backs this

Recorded sources

2 sources for · 0 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Evidence from clinical studies and animal models demonstrates that unilateral damage or transection of the vagus nerve produces specific physiological and anatomical deficits, such as ipsilateral laryngeal and palatal paralysis.

The analysis

The claim is specific, empirical, and contestable regarding whether unilateral vagus nerve damage causes distinct physiological deficits. Papers 4 and 11 provide direct clinical and experimental evidence that unilateral vagal injury results in specific functional deficits like vocal cord/laryngeal paralysis, pharyngeal dysfunction, and altered organ-specific regulation. No papers refute the claim.

Evidence for · 2

Regulation of Carcinogenesis by Sensory Neurons and Neuromediators.

Recorded source metadata

Erin N, Shurin GV, Baraldi JH, Shurin MR. Regulation of Carcinogenesis by Sensory Neurons and Neuromediators.. 2022. https://doi.org/10.3390/cancers14092333

Paper 4 notes that specific vagal denervation or vagotomy in animal models leads to targeted physiological changes such as increased tumor growth and metastasis in specific organs.

Source citation format

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More for · 1

Palatal adhesion: The treatment of unilateral palatal paralysis after high vagus nerve injury

Recorded source metadata

James L. Netterville, Scott Fortune, Steve Stanziale, Cheryl R. Billante. Palatal adhesion: The treatment of unilateral palatal paralysis after high vagus nerve injury. 2002. https://doi.org/10.1002/hed.10134

Paper 11 documents that unilateral proximal vagus nerve injury results in specific physiological deficits, including ipsilateral laryngeal paralysis, palatal and pharyngeal paralysis, and velopharyngeal incompetence.

Source citation format

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first checked01 Aug 2026

judged → SUPPORTED · 8401 Aug 2026

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