trustme.bro/r/…
✓ checked
trust me, bro:
here is the receipt.
the claim

Prenatal testosterone exposure is regulated by maternal physiology and fetal genetics

the verdict
SUPPORTED
the evidence backs this
Recorded sources
4 sources for · 0 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Evidence from clinical and animal studies shows that prenatal testosterone exposure is significantly shaped and regulated by maternal physiological factors as well as fetal and genetic characteristics.

The analysis

The claim states that prenatal testosterone exposure is regulated by maternal physiology and fetal genetics. Papers 0, 1, 5, and 10 provide robust evidence demonstrating that maternal factors (hormones, metabolism, and placental function) and fetal/genetic factors (such as steroidogenic genes and fetal sex differences) jointly regulate testosterone levels and action during gestation.

Evidence for · 4
Testosterone levels in midtrimester maternal and fetal plasma and amniotic fluid
Recorded source metadata

C. H. Rodeck, D. Gill, D. A. Rosenberg, W. P. Collins. Testosterone levels in midtrimester maternal and fetal plasma and amniotic fluid. 1985. https://doi.org/10.1002/pd.1970050303

Demonstrates differences in testosterone levels between maternal plasma, amniotic fluid, and fetal plasma, reflecting maternal and fetal physiological contributions.

See more details
More for · 3
Effect of Maternal Progesterone Exposure on Fetal Testosterone in Mice
Recorded source metadata

G. Pointis, M.T. Latreille, M.O. Richard, P. D’Athis, L. Cedard. Effect of Maternal Progesterone Exposure on Fetal Testosterone in Mice. 2009. https://doi.org/10.1159/000242005

Shows that maternal exposure to exogenous compounds like progesterone alters fetal testosterone levels, supporting maternal physiological regulation.

POLYCYSTIC OVARY SYNDROME: ORIGINS AND IMPLICATIONS: Gestational anti-Müllerian hormone and testosterone excess combined with maternal adiposity program for polycystic ovary syndrome.
Recorded source metadata

David H Abbott, Jon E Levine, Phillip A Dumesic, Vasantha Padmanabhan, Daniel A Dumesic. POLYCYSTIC OVARY SYNDROME: ORIGINS AND IMPLICATIONS: Gestational anti-Müllerian hormone and testosterone excess combined with maternal adiposity program for polycystic ovary syndrome.. 2025. https://doi.org/10.1530/REP-25-0120

Discusses how maternal metabolic traits and PCOS risk genes regulating androgen biosynthesis interact to influence fetal hormone exposure.

Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome in adulthood.
Recorded source metadata

Brooke Tata, Nour El Houda Mimouni, Anne-Laure Barbotin, Samuel A Malone, Anne Loyens, Pascal Pigny, Didier Dewailly, Sophie Catteau-Jonard, Inger Sundström-Poromaa, Terhi T Piltonen, Federica Dal Bello, Claudio Medana, Vincent Prevot, Jerome Clasadonte, Paolo Giacobini. Elevated prenatal anti-Müllerian hormone reprograms the fetus and induces polycystic ovary syndrome in adulthood.. 2018. https://doi.org/10.1038/s41591-018-0035-5

Indicates that maternal factors (such as elevated AMH during pregnancy) drive fetal testosterone excess and neuroendocrine programming.

The paper trail · every fact has a biography
first checked01 Aug 2026
judged → SUPPORTED · 8701 Aug 2026
Anyone with this link can read the claim and its public receipt, including any personal information in that text. Open permanent receipt.
Check your own claim
Challenge the receipt
Requests are recorded for review. This does not start an automatic check or guarantee a response time.
trust me, bro: win the argument, pass the class, survive peer review.

Citation formatting by citeproc-js (Frank Bennett) and the Citation Style Language project. Source and licenses.

This receipt is an automated verdict against our published method · not an opinion about any author or publication.
Terms · Privacy · How verdicts work · Dispute this receipt