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the claim

Magnesium supplementation causes colon cancer.

the verdict
INSUFFICIENT LEANING
Recorded sources
0 sources for · 4 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Is it true that magnesium supplementation causes colon cancer?

Unclear. The retrieved items offer only partial, indirect evidence on magnesium and colon cancer: an in-vitro study found a magnesium supplement reduced colon cancer cell viability, and a cell-line paper links magnesium deficiency (not supplementation) to a more aggressive colon cancer phenotype. No item directly tests whether magnesium supplementation causes colon cancer in humans.

Verdict: PARTIAL LEAN · 0 sources for, 4 against · Last checked

The private library was unavailable for some searches in this check. Missing library results are not evidence against the claim.

Evidence against · 4
Calcium, Magnesium, and Colorectal Cancer -
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Calcium, Magnesium, and Colorectal Cancer - PMC Lock. (n.d.). pmc.ncbi.nlm.nih.gov. https://doi.org/10.1097/EDE.0b013e31824deb09

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More against · 3
Investigating the potential selective effects of a Kiperin multi-component magnesium supplement on colon cancer and normal cells
2025
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Batur, L. K., Yavaş, C., & Polat, Y. (2025). Investigating the potential selective effects of a Kiperin multi-component magnesium supplement on colon cancer and normal cells. doi.org. https://doi.org/10.1038/s41598-025-32854-6

Magnesium deficiency with high calcium-to-magnesium ratio promotes a metastatic phenotype in the CT26 colon cancer cell line.
2020
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Kumar, G., Chatterjee, P., Madankumar, S., Mehdi, S. F., Xue, X., & Metz, C. (2020). Magnesium deficiency with high calcium-to-magnesium ratio promotes a metastatic phenotype in the CT26 colon cancer cell line.. doi.org. https://doi.org/10.1684/mrh.2020.0470

TRPM6 is Essential for Magnesium Uptake and Epithelial Cell Function in the Colon
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TRPM6 is Essential for Magnesium Uptake and Epithelial Cell Function in the Colon. (n.d.). cir.nii.ac.jp. https://doi.org/10.3390/nu10060784

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