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the claim

Influenza infection raises the risk of a secondary bacterial chest infection such as pneumonia.

the verdict
SUPPORTED
the evidence backs this
Recorded sources
4 sources for · 0 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Is it true that influenza infection raises the risk of a secondary bacterial chest infection such as pneumonia?

Yes. A reference article on influenza and a clinical review both state that influenza infection commonly progresses to pneumonia and that secondary bacterial infections during or after influenza are a frequent cause of pneumonia. Animal-model studies add supporting but non-human evidence.

Verdict: SUPPORTED · 4 sources for, 0 against · Last checked

The private library was unavailable for some searches in this check. Missing library results are not evidence against the claim.

Evidence for · 4
Influenza
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Influenza. (n.d.). en.wikipedia.org. https://en.wikipedia.org/wiki/Influenza

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More for · 3
Effect of triazavirine on the outcome of a lethal influenza infection and secondary bacterial pneumonia following influenza in mice
2018
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A., L. I., N., F. I., & R., M. N. (2018). Effect of triazavirine on the outcome of a lethal influenza infection and secondary bacterial pneumonia following influenza in mice. zenodo.org. https://doi.org/10.18527/2500-2236-2017-4-1-52-57

Nontypeable <i>Haemophilus influenzae</i> specific memory TH17 cells protect against secondary bacterial pneumonia following Influenza A virus infection
2023
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Yang, Y. (2023). Nontypeable <i>Haemophilus influenzae</i> specific memory TH17 cells protect against secondary bacterial pneumonia following Influenza A virus infection. doi.org. https://doi.org/10.4049/jimmunol.210.supp.158.05

Bench-to-bedside review: Bacterial pneumonia with influenza - pathogenesis and clinical implications -
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Bench-to-bedside review: Bacterial pneumonia with influenza - pathogenesis and clinical implications - PMC Lock. (n.d.). pmc.ncbi.nlm.nih.gov. https://doi.org/10.1186/cc8893

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