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the claim

Deficiencies in DNA repair are linked with human cancer.

the verdict
SUPPORTED
the evidence backs this
Recorded sources
8 sources for · 0 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Is it true that deficiencies in DNA repair are linked with human cancer?

Yes. Extensive scientific literature confirms that deficiencies in DNA repair pathways compromise genomic integrity and serve as fundamental drivers of human cancer initiation, progression, and predisposition.

Verdict: SUPPORTED · 8 sources for, 0 against · Last checked

The analysis

The retrieved papers overwhelmingly support the link between DNA repair deficiencies and various human cancers, detailing specific mechanisms like homologous recombination deficiency and mismatch repair defects across multiple tumor types.

Evidence for · 8
Molecular landscape and clinical translation of DNA damage response alterations in solid tumors: A pan-cancer perspective on precision oncology.
2026
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H, Z., J, S., Y, W., C, G., & J, L. (2026). Molecular landscape and clinical translation of DNA damage response alterations in solid tumors: A pan-cancer perspective on precision oncology.. doi.org. https://doi.org/10.1016/j.neo.2026.101315

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More for · 7
The Genetic Landscape of Colorectal Cancer: From Molecular Alterations to Therapeutic Decision Pathways.
2026
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CM, M., T, I., A, C., P, T., VR, M., & OC, F. (2026). The Genetic Landscape of Colorectal Cancer: From Molecular Alterations to Therapeutic Decision Pathways.. doi.org. https://doi.org/10.3390/cancers18152526

iPSC models of hereditary cancer predisposition: Mechanistic insights into premalignant evolution and lineage-specific vulnerability.
2026
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BB, G., QB, Y., WL, L., & N, X. (2026). iPSC models of hereditary cancer predisposition: Mechanistic insights into premalignant evolution and lineage-specific vulnerability.. doi.org. https://doi.org/10.1016/j.bbcan.2026.189695

The WHO Classification of Genetic Tumour Syndromes: Considerations for Genetics.
2026
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IA, C., MJ, A., JD, K., ERE, D., AJ, G., AJ, L., IM, F., SM, P., MA, R., KRM, L., R, D., EA, B., SE, P., A, H., MF, W., G, G., H, W., WD, F., & D, L. (2026). The WHO Classification of Genetic Tumour Syndromes: Considerations for Genetics.. doi.org. https://doi.org/10.1111/cge.70195

Breast-Ovarian Hereditary Cancer Syndrome: Beyond <i>BRCA1</i> and <i>BRCA2</i>.
2026
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E, I. & A, S. (2026). Breast-Ovarian Hereditary Cancer Syndrome: Beyond <i>BRCA1</i> and <i>BRCA2</i>.. doi.org. https://doi.org/10.3390/curroncol33090556

Distinct functions of mammalian RAD51 paralogs in genome maintenance.
2026
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HK, D., D, B., S, K., & G, N. (2026). Distinct functions of mammalian RAD51 paralogs in genome maintenance.. doi.org. https://doi.org/10.1042/bst20250440

Expanding the therapeutic scope of PARP inhibitors in breast cancer: current indications and future directions.
2026
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CS, W., A, R., PA, K., GI, S., SM, T., JE, G., & F, L. (2026). Expanding the therapeutic scope of PARP inhibitors in breast cancer: current indications and future directions.. doi.org. https://doi.org/10.1172/jci207632

From Germline Susceptibility to Therapeutic Vulnerability: DNA Damage Response Gene Mutations Driving Multiple Myeloma Evolution and Precision Therapy.
2026
Get citation
Q, S., Y, W., L, C., & J, Q. (2026). From Germline Susceptibility to Therapeutic Vulnerability: DNA Damage Response Gene Mutations Driving Multiple Myeloma Evolution and Precision Therapy.. doi.org. https://doi.org/10.1155/humu/7757115

What does the evidence say?

The retrieved papers overwhelmingly support the link between DNA repair deficiencies and various human cancers, detailing specific mechanisms like homologous recombination deficiency and mismatch repair defects across multiple tumor types.

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