trustmebro.to/r/…
✓ checked
trust me, bro:
here is the receipt.
the claim

Cancer occurs when cells multiply without stopping and old cells do not die when they should, allowing them to spread into surrounding tissues.

the verdict
SUPPORTED
the evidence backs this
Recorded sources
4 sources for · 0 against

Counts group repeated records of the same source within each side. They do not measure evidence strength or source independence.

Is it true that cancer occurs when cells multiply without stopping and old cells do not die when they should, allowing them to spread into surrounding tissues?

Yes. The retrieved scientific literature strongly supports the foundational oncology definition that cancer involves uncontrolled cell multiplication and the evasion of programmed cell death (apoptosis).

Verdict: SUPPORTED · 4 sources for, 0 against · Last checked

The analysis

The claim outlines the core hallmarks of cancer: continuous cellular proliferation and evasion of apoptosis. Multiple retrieved reviews and experimental papers (such as [1], [2], [4], and [8]) explicitly describe how cancer cells evade cell death and undergo abnormal, unrestrained multiplication, aligning perfectly with the claim.

Evidence for · 4
REG4 promotes the proliferation and anti-apoptosis of cancer
2022
The paper discusses how REG4 promotes cancer cell proliferation and inhibits apoptosis.
Get citation
Zheng, H., Xue, H., & Zhang, C. (2022). REG4 promotes the proliferation and anti-apoptosis of cancer. doi.org. https://doi.org/10.3389/fcell.2022.1012193

See more details
More for · 3
Promotion of BZW2 by LINC00174 through miR-4500 inhibition enhances proliferation and apoptosis evasion in laryngeal papilloma
2020
The study demonstrates that regulating cell proliferation and apoptosis evasion drives tumor development.
Get citation
Liu, J., Yang, T., Zhang, Y., & Wang, S. (2020). Promotion of BZW2 by LINC00174 through miR-4500 inhibition enhances proliferation and apoptosis evasion in laryngeal papilloma. doi.org. https://doi.org/10.1186/s12935-020-01559-3

MicroRNA Dysregulation in HPV-Driven Cervical Cancer: A Review of Oncoprotein-Targeted Signaling Pathways.
2026
The review highlights sustained proliferation and the evasion of apoptosis as key oncogenic processes in cancer.
Get citation
GM, P., S, C., PRD, S., AJA, F., SMJ, B., RM, R., MMC, M., & RL, C. (2026). MicroRNA Dysregulation in HPV-Driven Cervical Cancer: A Review of Oncoprotein-Targeted Signaling Pathways.. doi.org. https://doi.org/10.3390/life16081332

B-MYB (MYBL2): from cell cycle regulator to an oncogenic player.
2026
The review notes that overexpression of B-MYB leads to uncontrolled proliferation and apoptosis evasion.
Get citation
A, M., PA, O., JL, M., & C, C. (2026). B-MYB (MYBL2): from cell cycle regulator to an oncogenic player.. doi.org. https://doi.org/10.1016/j.bbcan.2026.189697

What does the evidence say?

The claim outlines the core hallmarks of cancer: continuous cellular proliferation and evasion of apoptosis. Multiple retrieved reviews and experimental papers (such as [1], [2], [4], and [8]) explicitly describe how cancer cells evade cell death and undergo abnormal, unrestrained multiplication, aligning perfectly with the claim.

Receipt History
Anyone with this link can read the claim and its public receipt, including any personal information in that text. Open permanent receipt.
Check your own claim
Challenge the receipt
Requests are recorded for review. This does not start an automatic check or guarantee a response time.

See the evidence timeline

Citation formatting by citeproc-js (Frank Bennett) and the Citation Style Language project. Source and licenses.

This receipt is an automated verdict against our published method · not an opinion about any author or publication.
Terms · Privacy · How verdicts work