Viruses are not self-propelled entities; instead, they completely depend on host cell machinery, cytoskeletal networks, and hijacked cellular pathways to facilitate their transport and replication.
Evidence against · 10
Influenza A Virus Cell Entry, Replication, Virion Assembly and Movement.
2018 · cited by 0
Influenza viruses rely entirely on host cellular mechanisms and internal networks to coordinate the transport of their components in and out of the cell.
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More against · 9
Vaccinia virus motility.
2003 · cited by 0
Vaccinia virus depends on host microtubules and actin polymerization rather than self-generated propulsion for its intracellular movement.
Actin-based motility of intracellular pathogens.
2005 · cited by 0
Viruses such as vaccinia exploit host actin cytoskeletons and the Arp2/3 complex to facilitate movement rather than propelling themselves independently.
Nuclear Cytoskeleton in Virus Infection.
2022 · cited by 0
Viruses rely on host nuclear cytoskeleton and cellular kinases for trafficking and nuclear egress.
Similarities in intracellular transport of plant viral movement proteins BMB2 and TGB3.
2017 · cited by 0
Plant viral movement proteins exploit the host ER-actin network for intracellular transport rather than driving themselves.
Actin-based motility of intracellular microbial pathogens.
2001 · cited by 0
Microbial pathogens utilize preexisting mammalian pathways of actin rearrangement to induce movement rather than intrinsic propulsion.
Viruses That Exploit Actin-Based Motility for Their Replication and Spread.
2017 · cited by 0
Viruses depend entirely on host cells and manipulate the host actin network for their replication and spread.
Arp2/3-mediated actin-based motility: a tail of pathogen abuse.
2013 · cited by 0
Viruses evolve strategies to harness host actin polymerization rather than possessing intrinsic self-propulsion machinery.
Actin-based motility of vaccinia virus.
1995 · cited by 0
Vaccinia virus induces host actin tails to be propelled by host cellular machinery rather than intrinsic movement.
Viruses that ride on the coat-tails of actin nucleation.
2015 · cited by 0
Viral pathogens recruit and activate cellular actin nucleators to provide the mechanical force needed for transport.