We explore how discreet input can be provided using the tensor tympani - a small muscle in the middle ear that some people can voluntarily contract to induce a dull rumbling sound. We investigate the prevalence and ability to control the muscle through an online questionnaire (N=192) in which 43.2% of respondents reported the ability to “ear rumble”. Data collected from participants (N=16) shows how in-ear barometry can be used to detect voluntary tensor tympani contraction in the sealed ear canal. This data was used to train a classifier based on three simple ear rumble “gestures” which achieved 95% accuracy. Finally, we evaluate the use of ear rumbling for interaction, grounded in three manual, dual-task application scenarios (N=8). This highlights the applicability of EarRumble as a low-effort and discreet eyes- and hands-free interaction technique that users found “magical” and “almost telepathic”.

BACKGROUND: The tensor tympani muscle is the largest muscle within the middle ear. Its voluntary contraction is a very unusual event. Only a few papers have documented its audiometric effects. OBJECTIVE: To report an unusual case of voluntary tensor tympani muscle contraction and describe its audiometric effects. CASE REPORT: A 27-year-old man, who presented complaining of voluntarily evoked bilateral tinnitus, was found to be able to voluntarily contract the tensor tympani muscle in both ears simultaneously. Audiograms were performed under conditions of rest and maximal contraction of the tensor tympani muscle. The most remarkable effects were conductive hearing loss at lower frequencies and an increase in middle-ear impedance. CONCLUSION: The importance of the tensor tympani muscle in middle-ear physiology remains unclear. It has been related to the attenuation of sounds produced during the mastication process. Voluntary control over the tensor tympani muscle is an extremely rare event. However, an understanding of the potential audiometric effects of its contraction could aid the diagnosis of hearing disorders.

<h4>Background</h4>Tensor tympani contraction may have a "signature" audiogram. This study demonstrates audiometric findings during voluntary tensor tympani contraction.<h4>Methods</h4>Five volunteers possessing the ability to voluntarily contract their tensor tympani muscles were identified and enrolled. Tensor tympani contraction was confirmed with characteristic tympanometry findings. Study subjects underwent conventional audiometry. Air conduction and bone conduction threshold testing was performed with and without voluntary tensor tympani contraction.<h4>Main outcome measure</h4>Changes in air conduction and bone conduction thresholds during voluntary tensor tympani contraction.<h4>Results</h4>Audiometric results demonstrate a low frequency mixed hearing loss resulting from tensor tympani contraction. Specifically, at 250 Hz, air conduction thresholds increased by 22 dB and bone conduction thresholds increased by 10 dB.<h4>Conclusions</h4>Previous research has demonstrated a low frequency conductive hearing loss in the setting of tensor tympanic contraction. This is the first study to demonstrate a low frequency mixed hearing loss associated with tensor tympani contraction. This finding may aid in the diagnosis of disorders resulting from abnormal tensor tympani function. Tensor tympani contraction should be included on the differential for low frequency mixed hearing loss.

Loss of communication with loved ones and carers is one of the most isolating and debilitating effects of many neurological disorders. Assistive technology (AT) supports individuals with communication, but the acceptability of AT solutions is highly variable. In this paper a novel ear based control method of AT, the concept of ’EarSwitch’, is presented. This new approach is based on detecting ear rumbling, which is the voluntary contraction of the tensor tympani muscle (TTM), resulting in observable movement of the eardrum and a dull rumbling sound. ’EarSwitch’ has the potential to be a discreet method that can complement existing AT control methods. However, only a subset of the population can ear rumble and little is known about the ability of rumbling in populations with neurological disorders. To explore the viability of the ’EarSwitch’ concept as an AT control method we conducted in-depth online surveys with (N=1853) respondents from the general population and (N=170) respondents with self-declared neurological disorders including Motor Neurone Disease (MND) and Multiple Sclerosis (MS).This is the largest ever study to explore ear rumbling and the first to explore whether rumbling is preserved among individuals with neurological disorders. In addition, we validated rumbling, and investigated usability of the ’EarSwitch’ concept as a control input, using in-person otoscopic examination with a subset of participants. A significant proportion of the population with neurological disorders could benefit from ’EarSwitch’ controllable AT. The upper bound prevalence of the ability to rumble without accompanying movements was 55% in the general population, 38% in the neurological population, and 20% of participants with MND (N=95) reported this ability. During the validation procedure, participants achieved high accuracy in self-reporting the ability to rumble (80%) and proved concept of using the ’EarSwitch’ method to control a basic interface. ’EarSwitch’ is a potential new AT control method control, either by itself or as a supplement to other existing methods. Results demonstrate self-reported ear rumbling is present among patients with different neurological disorders, including MND. Further research should explore how well the ability to rumble is preserved in different types and stages of neurological disorders.

The tensor tympani is a skeletal muscle within the middle ear, located in the bony canal above the bony part of the auditory tube, and connects to the The tensor tympani is a skeletal muscle within the middle ear, located in the bony canal above the bony part of the auditory tube, and connects to the malleus bone. Its role is to dampen loud sounds, such as those produced from chewing, shouting, or thunder. Because its reaction time is not fast enough, the muscle cannot protect against hearing damage caused by sudden loud sounds, like explosions The tensor tympani is a skeletal muscle within the middle ear, located in the bony canal above the bony part of the auditory tube, and connects to the malleus bone. Its role is to dampen loud sounds, such as those produced from chewing, shouting, or thunder. Because its reaction time is not fast enough, the muscle cannot protect against hearing damage caused by sudden loud sounds, like explosions or gunshots, however some individuals have voluntary control over the muscle, and may tense it pre-emptively. The tensor tympani is a muscle that is present in the middle ear. It arises from the cartilaginous part of the auditory tube, and the adjacent great wing of the sphenoid. It then passes through its own canal, and ends in the tympanic cavity as a slim tendon that connects to the handle of the malleus. The tendon makes a sharp bend around the processus cochleariformis, part of the wall of its cavity, before it joins with the malleus. The tensor tympani receives blood from the middle meningeal artery via the superior tympanic branch. It is one of two muscles in the tympanic cavity, the other being the stapedius. Hearing Middle ear Ossicles Stapedius – the other major muscle in the middle ear Acoustic reflex Hyperacusis

can voluntarily produce this rumbling sound by contracting the tensor tympani muscle of the middle ear. The rumbling sound can also be heard when the neck Skeletal muscle (commonly referred to as muscle) is one of the three types of vertebrate muscle tissue, the others being cardiac muscle and smooth muscle. They are part of the musculoskeletal system, and typically are attached by tendons to the bones of a skeleton. The skeletal muscle cells (myocytes) are much longer than in the other types of muscle tissue, and are also known as muscle fibers. T Mu… Neuromuscular diseases affect the muscles and their nervous control. In general, problems with nervous control can cause spasticity or paralysis, depending on the location and nature of the problem. A number of movement disorders are caused by neurological disorders such as Parkinson's disease and Huntington's disease where there is central nervous system dysfunction. Symptoms of muscle diseases may include weakness, spasticity, myoclonus and myalgia. Diagnostic procedures that may reveal muscular disorders include testing creatine kinase levels in the blood and electromyography (measuring electrical activity in muscles). In some cases, muscle biopsy may be done to identify a myopathy, as well as genetic testing to identify DNA abnormalities associated with specific myopathies and dystrophies. A non-invasive elastography technique that measures muscle noise is undergoing experimentation to provide a way of monitoring neuromuscular disease. The sound…